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Axonal transport and secretion of fibrillar forms of α-synuclein, Aβ42 peptide and HTTExon 1.

Archive ouverte | Brahic, Michel | CCSD

International audience. Accruing evidence suggests that prion-like behavior of fibrillar forms of α-synuclein, β-amyloid peptide and mutant huntingtin are responsible for the spread of the lesions that characterize ...

Neuron-to-neuron transmission of α-synuclein fibrils through axonal transport.

Archive ouverte | Freundt, Eric C | CCSD

International audience. The lesions of Parkinson disease spread through the brain in a characteristic pattern that corresponds to axonal projections. Previous observations suggest that misfolded α-synuclein could be...

ERAD defects and the HFE-H63D variant are associated with increased risk of liver damages in Alpha 1-Antitrypsin Deficiency

Archive ouverte | Joly, Philippe | CCSD

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