Two avirulence genes of Leptosphaeria maculans, displaying suppressive interactions, share a common structural pattern and are part of a larger effector family

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Lazar, Noureddine | Petit-Houdenot, Yohann | Talbi, Nacera | Li de la Sierra-Gallay, Inès | Emilie, Zelie | Blondeau, Karine | van de Wouw, Angela | Ollivier, Bénédicte | Mesarich, Carl H. | Idnurm, Alexander | van Tilbeurgh, Herman | Fudal, Isabelle

Edité par CCSD -

International audience. Recognition of a pathogen avirulence (AVR) protein triggers a set of immune responses grouped under the term Effector-Triggered Immunity (ETI). Pathogens can escape ETI by different mechanisms including complete deletion or down-regulation of the AVR genes, point mutations allowing recognition to be evaded while maintaining the virulence function of the AVR protein, and acquisition of new effectors able to suppress recognition. In some cases, the effector that suppresses ETI can itself be recognized by an R gene. A few examples of such mechanism were recently described in fungi but the underlying mechanisms have remained unexplained.

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