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Host-Derived Lipids from Tuberculous Pleurisy Impair Macrophage Microbicidal-Associated Metabolic Activity

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Marín Franco, José Luis | Genoula, Melanie | Corral, Dan | Duette, Gabriel | Ferreyra, Malena | Maio, Mariano | Dolotowicz, María Belén | Aparicio-Trejo, Omar Emiliano | Patiño-Martínez, Eduardo | Charton, Alison | Métais, Arnaud | Fuentes, Federico | Soldan, Vanessa | Moraña, Eduardo José | Palmero, Domingo | Ostrowski, Matías | Schierloh, Pablo | Sánchez-Torres, Carmen | Hernández-Pando, Rogelio | Pedraza-Chaverri, José | Rombouts, Yoann | Hudrisier, Denis | Layre, Emilie | Vérollet, Christel | Maridonneau-Parini, Isabelle | Neyrolles, Olivier | Sasiain, María del Carmen | Lugo-Villarino, Geanncarlo | Balboa, Luciana | Marín Franco, José, Luis | Dolotowicz, María, Belén | Aparicio-Trejo, Omar, Emiliano | Moraña, Eduardo, José | Sasiain, María, del Carmen

Edité par CCSD ; Elsevier Inc -

International audience. Highlights d Tuberculous pleural effusion (TB-PE) shifts glycolysis to OXPHOS in M1 macrophages d Metabolic shift is accompanied by low HIF-1a activity and high M. tuberculosis burden d HIF-1a stabilization reverts the metabolic shift and inhibitory effects of TB-PE d Host-derived lipids (eicosanoid-enriched fraction) in TB-PE drive the metabolic shift

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